期刊名称：ePlasty: Open Access Journal of Plastic and Reconstructive Surgery
印刷版ISSN：1937-5719
出版年度：2017
卷号：17
语种：English
出版社：Open Science Co. LLC
摘要：DESCRIPTIONA 5-year old boy presented to the pediatric plastic surgery clinic for hypernasal speech. He was born with a cleft palate and underwent repair at 11 months of age. A short velum was noted (Fig 1), so he underwent palatal lengthening with double-opposing buccal flaps. Postoperatively, his hypernasality was corrected.QUESTIONSWhat is velopharyngeal dysfunction (VPD)?How is velopharyngeal insufficiency (VPI) diagnosed?What treatment options are available for VPI?What is the palatal lengthening with double-opposing buccal flaps procedure?DISCUSSIONVelopharyngeal dysfunction is defined as the inability of the velopharyngeal valve (VPV) to close properly.1It manifests as altered speech and nasal regurgitation. Velopharyngeal dysfunction can be classified into 3 main categories based on the underlying cause. Velopharyngeal incompetence is due to a neurological or neuromuscular cause leading to impaired function of the VPV despite normal anatomy. Velopharyngeal mislearning is a less common form of VPD, in which function and anatomy of the velopharyngeal (VP) mechanism are intact, but the patient has mislearned how to produce certain speech sounds accurately. Velopharyngeal insufficiency represents an inadequate closure of the VPV due to anatomical or structural abnormalities. It can develop secondary to surgical intervention, congenital malformations, or trauma. The most common congenital causes of VPI include cleft palate and velopharyngeal disproportion. Surgical procedures such as palatoplasty, tumor resection, and adenotonsillectomy can also result in VPI.2Velopharyngeal insufficiency can be diagnosed through auditory and instrumental methods, with both methods frequently utilized to obtain an accurate diagnosis.3Perceptual speech evaluation allows articulation, resonance, nasal airway emission, and voice to be assessed.4,5Video fluoroscopy and nasendoscopy are the most common imaging modalities used in the diagnosis of VPI.5These methods provide direct, dynamic images of the anatomy and function of the VPV in vivo.3They can assess closure patterns, gaps, and anatomical variations, which are useful in both identifying the etiology of dysfunction and surgical planning.2Nonsurgical treatment options for VPI include speech therapy and prosthetics. Speech therapy remains the first-line treatment of nearly all patients with VPI.3As an isolated therapy, it has the highest success in patients with mild insufficiency. When surgical intervention is necessary, speech therapy almost always accompanies the procedure. Obturators and prostheses are most useful in patients in whom surgery is not an option.2The goal of surgical intervention is to narrow the nasopharynx and restore VPV competence without causing hyponasality or upper airway obstruction.4The Furlow: double-opposing Z-plasty palatoplasty reconstructs the levator sling by repositioning the posterior myomucosal flap. This returns the levator musculature to the appropriate horizontal physiological position in addition to lengthening the velum.3,5In a posterior pharyngeal flap, the soft palate is surgically adhered to a superiorly based flap from the posterior pharyngeal wall, creating a static structure that reduces the size of the velopharyngeal opening in the sagittal plane. This repair may be more successful in VPD with intact lateral pharyngeal wall motion. The sphincter pharyngoplasty utilizes bilateral palatopharyngeal flaps from the posterolateral pharyngeal walls. They are rotated posterior and medially and connected with the posterior pharyngeal wall with a joining incision. This procedure may be better for patients with poor VPV motion in the coronal plane.2,3Palatal lengthening with bilateral opposed buccal flaps (Figs 2aand2b) is designed to correct VPI by allowing the velum to move posterior and improving the closure of the VP sphincter.6In this procedure, the velum is released from the hard palate with a transverse incision. Abnormal muscle attachments are released, and the soft palate falls posteriorly into a more anatomical position. Bilateral buccal myomucosal flaps based at the retromolar trigone are then raised to fill the soft tissue gap between the hard and soft palates (Figs 3and4). The 2 flaps are opposed together, with the mucosa of the flaps reconstructing the nasal and oral sides of the palatal defect. This approach avoids surgical alteration of the velar muscles, decreasing potential scarring and leading to excellent speech outcomes.6CONCLUSIONVelopharyngeal insufficiency is commonly seen in patients with cleft palate. Many techniques have been described to improve VP function and speech. The palatal lengthening procedure has been shown to be a safe procedure with minimal donor site morbidity and no postoperative sleep apnea.
