期刊名称：General Internal Medicine and Clinical Innovations
电子版ISSN：2397-5237
出版年度：2020
卷号：5
期号：1
页码：1-3
DOI：10.15761-GIMCI.1000183
语种：English
出版社：Open Access Text
摘要：Renal handling of magnesium (Mg) is adaptive mechanism, but it’s affected when renal function declines. In moderate chronic kidney disease (CKD), loss of glomerular filtration is compensated by higher fractional excretion of Mg. In more advanced CKD however, (creatinine clearance st group (32 CKD diabetic patients); 2nd group (30 CKD patients without diabetes). Biochemical determinations were made, and the estimated glomerular filtration rate (GFR) was measured. Bone mineral density (BMD) was measured by dual-energy X-ray absorptiometry. sMg was inversely correlated with sCa (Pst group (diabetic). The CKD diabetic patients had lower serum albumin (ALB) and a higher hypomagnesemia and osteoporosis than the nondiabetic patients. sMg inversely correlated with e-GFR in the CKD patients in both groups. sMg showed higher correlation with 25-hydroxyvitamin D in 2nd non-diabetic group.
关键词：magnesium (Mg); chronic kidney disease (CKD); diabetic patients; bone mineral metabolism (BMM)
