摘要：A high degree of resistance to cyclodiene insecticides, which appeared in a previously susceptible housefly strain maintained without exposure to insecticides but propagated from early-emerging adults to increase susceptibility to DDT, was found to be due to a single autosomal factor. Subsequent selection of a substrain for late adult emergence over 50 generations was unsuccessful in materially reversing the dieldrin-resistance or in demonstrating that selection of early-emerging flies was responsible for its appearance. However, selection and propagation of the knockdown-susceptible fraction of the population with lindane over 30 generations eliminated cyclodiene-resistance entirely. This method can be of value in maintaining laboratory strains at a normal level of susceptibility to cyclodiene insecticides. It is apparent, nevertheless, that various manipulations of a standard strain may affect its toxicological as well as other characteristics. The authors stress that when a standard reference strain is required for an extended period of time, it should be rigorously controlled and continuously evaluated. Full text Full text is available as a scanned copy of the original print version. Get a printable copy (PDF file) of the complete article (1.2M), or click on a page image below to browse page by page. Links to PubMed are also available for Selected References . 167 168 169 170 171 172 173 174 175 176
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