期刊名称：Proceedings of the National Academy of Sciences
印刷版ISSN：0027-8424
电子版ISSN：1091-6490
出版年度：2019
卷号：116
期号：27
页码：13173-13173
DOI：10.1073/pnas.1907923116
出版社：The National Academy of Sciences of the United States of America
摘要：As the mathematical model developed by Woller et al. (1) also considers the role of SIRT1 in circadian clock, we regret not citing this work. However, we feel the inferences drawn in Furlan et al.’s (2) letter conflate 2 distinct studies with disparate purposes and methods. In Foteinou et al. (3), we address the role of SIRT1 in cell-autonomous clock function. We undertook this experimentation and modeling to resolve the conflicting reports by Asher et al. (4) and Nakahata et al. (5), elucidating … [↵][1]5To whom correspondence may be addressed.
