摘要：Since the discovery of the first weed species,Senecio vulgaris L,resistant to triazines,a total of 415 biotypes of weed species have been determined that are resistant to herbicides,i.e.183 weed species,of which 110 are dicotyledonous and 73 grass species,belonging to 18 groups with different mechanisms of action.Rapid increase in the number of these populations’ representatives continues steadily through frequent use of the same herbicides or those with identical mechanism of action.In plants,resistance can naturally spread,or it can be induced by techniques such as genetic engineering or selection of resistant biotypes obtained by tissue culture or mutagenesis.In respect to tolerant plants,resistant biotypes most frequently do not only survive commercial herbicide rates,but extremely high rates as well.Resistant biotypes with multifold resistance mechanisms represent a challenge for their control.Researchers keep trying to develop easier methods of detecting changes in the target sites of resistance mechanisms by using molecular biology.
其他摘要：Od prve rezistentne korovske vrste Senecio vulgaris L.na triazinske herbicide do danas,utvrđeno je 415 biotipova korovskih vrsta rezistentnih na herbicide iz 18 grupa različitih mehanizama delovanja,kod 183 korovske vrste,od kojih 110 dikotiledonih i 73 travna.Rapidno povećavanje broj predstavnika ovih populacija se nastavlja iz dana u dan,učestalom upotrebom istih ili herbicida istog mehanizma delovanja.U biljkama,rezistentnost se može prirodno raširiti ili može biti indukovana tehnikama kakve su genetički inžinjering ili selekcija rezistentnih biotipova dobijenih kulturom tkiva ili mutagenezom.Za razliku od tolerantnih biljaka,rezistentni biotipovi najčešće ne preživljavaju samo komercijalne količine herbicida nego i izuzetno velike količine.Rezistentni biotipovi sa višestrukim mehanizmima rezistentnosti predstavljaju izazov za suzbijanje.Istraživači pokušavaju molekularnom biologijom da olakšaju utvrđivanje promene ciljnog mesta kao mehanizma rezistentnosti.
