首页    期刊浏览 2024年11月25日 星期一
登录注册

文章基本信息

  • 标题:Role of Inflammatory Marker Interleukin 6 (IL-6) and Insulin in Diabetes and Diabetic Neuropathy
  • 作者:SV Joshi ; SR Tambwekar ; K Khadalia
  • 期刊名称:Bombay Hospital Journal
  • 印刷版ISSN:0524-0182
  • 出版年度:2008
  • 卷号:50
  • 期号:03
  • 出版社:Bombay Hospital Institute of Medical Sciences
  • 摘要:

    Introduction : Circulating IL-6 levels have been reported to be elevated in subjects with type 2
    diabetes. Diabetic peripheral neuropathy is a common complication of diabetes in 66% of type
    I and 59% of type 2 diabetics. 1 Diabetic polyneuropathy is the most commonly acquired diffuse
    disorder of the peripheral nervous system.2
    Aim : This study is focussed on the correlation between serum Interleukin-6 (IL-6), serum
    insulin and glycaemic status.
    Methods and Patients : Twenty-three subjects were selected for the study. Out of the total
    subjects, 17 were with type 2 diabetes mellitus and 6 with diabetic neuropathy. Two subjects
    with diabetic neuropathy had given consent for neurolysis. Blood sugar level was estimated by
    standard method. Serum IL-6 and insulin levels were measured by ELISA method. Attempts
    were made to elucidate the relationship between glycaemic status (or insulinaemia) and with
    diabetic neuropathy and neurolysis.
    Results : Results show that there is no direct relationship between fasting blood sugar and IL-
    6 however, raised IL-6 was observed in 66.66%) diabetic and in 50% with diabetic neuropathy. It
    was observed that though patients with diabetes were within glycaemic control raised IL-6
    levels revealed the presence of inflammation. Such patients should be followed up for early
    detection of neuropathy.
    Conclusion : Raised IL-6 levels in diabetics with glycaemic control revealed the presence of
    inflammation. Such patients should be followed up further to prevent neural damage

  • 关键词:Diabetic Neuropathy;Insulin;Inflammatory Markers;Interleukin 6;Diabetic Peripheral Neuropathy
Loading...
联系我们|关于我们|网站声明
国家哲学社会科学文献中心版权所有