1.Endothelial cells are activated by cytokine treatment to kill an intravascular parasite, Schistosoma mansoni, through the production of nitric oxide.

作　　者：I P Oswald ;I Eltoum ;T A Wynn 等

出　　处：Proceedings of the National Academy of Sciences. 1994 ;91(3):999-1003.doi:10.1073/pnas.91.3.999

出版社：The National Academy of Sciences of the United States of America

文章 ID：75182596

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2.Characterization of a programmed alteration in an 18S ribosomal gene that accompanies the experimental induction of drug resistance in Schistosoma mansoni

作　　者：P J Brindley ;S Heath ;A P Waters 等

出　　处：Proceedings of the National Academy of Sciences. 1991 ;88(17):7754-7758.doi:10.1073/pnas.88.17.7754

出版社：The National Academy of Sciences of the United States of America

文章 ID：75181664

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3.Major histocompatibility complex products restrict the adherence of cytolytic T lymphocytes to minor histocompatibility antigens or to trinitrophenyl determinants on schistosomula of Schistosoma mansoni

作　　者：M A Vadas ;A E Butterworth ;S Burakoff 等

出　　处：Proceedings of the National Academy of Sciences. 1979 ;76(4):1982-1985.doi:10.1073/pnas.76.4.1982

出版社：The National Academy of Sciences of the United States of America

文章 ID：200595478

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4.Enzymatic treatment transforms trypomastigotes of Trypanosoma cruzi into activators of alternative complement pathway and potentiates their uptake by macrophages

作　　者：T L Kipnis ;J R David ;C A Alper 等

出　　处：Proceedings of the National Academy of Sciences. 1981 ;78(1):602-605.doi:10.1073/pnas.78.1.602

出版社：The National Academy of Sciences of the United States of America

文章 ID：200597513

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5.Interleukin 10 inhibits macrophage microbicidal activity by blocking the endogenous production of tumor necrosis factor alpha required as a costimulatory factor for interferon gamma-induced activation.

作　　者：I P Oswald ;T A Wynn ;A Sher 等

出　　处：Proceedings of the National Academy of Sciences. 1992 ;89(18):8676-8680.doi:10.1073/pnas.89.18.8676

出版社：The National Academy of Sciences of the United States of America

文章 ID：200600827

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6.Helminth infection results in decreased virus-specific CD8+ cytotoxic T-cell and Th1 cytokine responses as well as delayed virus clearance.

作　　者：J K Actor ;M Shirai ;M C Kullberg 等

出　　处：Proceedings of the National Academy of Sciences. 1993 ;90(3):948-952.doi:10.1073/pnas.90.3.948

出版社：The National Academy of Sciences of the United States of America

文章 ID：200601859

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	A Sher	6篇
	I P Oswald	2篇
	S L James	2篇
	T A Wynn	2篇
	A E Butterworth	1篇

	A P Waters	1篇
	B Schwartz	1篇
	C A Alper	1篇
	D Paulin	1篇
	I Eltoum	1篇
	J A Berzofsky	1篇
	J K Actor	1篇
	J R David	1篇
	M A Vadas	1篇
	M C Kullberg	1篇
	M Shirai	1篇
	P Caspar	1篇
	P J Brindley	1篇
	R M Buller	1篇
	S Burakoff	1篇
	S Heath	1篇
	T F McCutchan	1篇
	T L Kipnis	1篇
	W D da Silva	1篇

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