1.A plasma lipoprotein containing only apolipoprotein E and with gamma mobility on electrophoresis releases cholesterol from cells.

作　　者：Y Huang ;A von Eckardstein ;S Wu 等

出　　处：Proceedings of the National Academy of Sciences. 1994 ;91(5):1834-1838.doi:10.1073/pnas.91.5.1834

出版社：The National Academy of Sciences of the United States of America

文章 ID：75182768

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2.Heterozygosity for apolipoprotein A-I(R160L)Oslo is associated with low levels of high density lipoprotein cholesterol and HDL-subclass LpA-I/A-II but normal levels of HDL-subclass LpA-I.

作　　者：T P Leren ;K S Bakken ;U Daum 等

出　　处：JLR Papers In Press. 1997 ;38(1):121-131.

出版社：American Society for Biochemistry and Molecular Biology

文章 ID：192687996

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3.A natural apolipoprotein A-I variant, apoA-I (L141R)Pisa, interferes with the formation of alpha-high density lipoproteins (HDL) but not with the formation of pre beta 1-HDL and influences efflux of cholesterol into plasma.

作　　者：R Miccoli ;Y Zhu ;U Daum 等

出　　处：JLR Papers In Press. 1997 ;38(6):1242-1253.

出版社：American Society for Biochemistry and Molecular Biology

文章 ID：192688163

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4.Structural and functional properties of reconstituted high density lipoprotein discs prepared with six apolipoprotein A-I variants.

作　　者：A Jonas ;A von Eckardstein ;K E Kézdy 等

出　　处：JLR Papers In Press. 1991 ;32(1):97-106.

出版社：American Society for Biochemistry and Molecular Biology

文章 ID：192686665

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5.Site-specific methionine sulfoxide formation is the structural basis of chromatographic heterogeneity of apolipoproteins A-I, C-II, and C-III.

作　　者：A von Eckardstein ;M Walter ;H Holz 等

出　　处：JLR Papers In Press. 1991 ;32(9):1465-1476.

出版社：American Society for Biochemistry and Molecular Biology

文章 ID：192686849

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6.Electrophoretic screening for genetic variation in apolipoprotein C-III: identification of a novel apoC-III variant, apoC-III(Asp45-->Asn), in a Turkish patient.

作　　者：S Lüttmann ;A von Eckardstein ;W Wei 等

出　　处：JLR Papers In Press. 1994 ;35(8):1431-1440.

出版社：American Society for Biochemistry and Molecular Biology

文章 ID：192687450

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7.A molecular defect causing fish eye disease: an amino acid exchange in lecithin-cholesterol acyltransferase (LCAT) leads to the selective loss of alpha-LCAT activity

作　　者：H Funke ;A von Eckardstein ;P H Pritchard 等

出　　处：Proceedings of the National Academy of Sciences. 1991 ;88(11):4855-4859.doi:10.1073/pnas.88.11.4855

出版社：The National Academy of Sciences of the United States of America

文章 ID：200599571

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	A von Eckardstein	7篇
	G Assmann	7篇
	H Funke	2篇
	U Daum	2篇
	Y Huang	2篇

	A Benninghoven	1篇
	A Jonas	1篇
	A Steinmetz	1篇
	C Droste	1篇
	E Köhler	1篇
	H Holz	1篇
	J J Albers	1篇
	J J Kastelein	1篇
	J Wessling	1篇
	K Berg	1篇
	K E Kézdy	1篇
	K S Bakken	1篇
	L Ose	1篇
	M Walter	1篇
	N Maeda	1篇
	P H Pritchard	1篇
	R Miccoli	1篇
	R Navalesi	1篇
	R W Mahley	1篇
	S Lüttmann	1篇
	S Wu	1篇
	T P Leren	1篇
	W Wei	1篇
	Y Zhu	1篇

	心理学	5篇
	自然科学总论	2篇

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	JLR Papers In Press	5篇
	Proceedings of the National Academy of Sciences	2篇

	1997	2篇
	1994	2篇
	1991	3篇