1.Origin of myofibroblasts in the fibrotic liver in mice

作　　者：Keiko Iwaisako ;Chunyan Jiang ;Mingjun Zhang 等

出　　处：Proceedings of the National Academy of Sciences. 2014 ;111(32):E3297-E3305.doi:10.1073/pnas.1400062111

出版社：The National Academy of Sciences of the United States of America

文章 ID：92927913

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2.Pleckstrin homology domain leucine-rich repeat protein phosphatases set the amplitude of receptor tyrosine kinase output

作　　者：Gloria Reyes ;Matt Niederst ;Ksenya Cohen-Katsenelson 等

出　　处：Proceedings of the National Academy of Sciences. 2014 ;111(38):E3957-E3965.doi:10.1073/pnas.1404221111

出版社：The National Academy of Sciences of the United States of America

文章 ID：93494936

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3.Conserved role for autophagy in Rho1-mediated cortical remodeling and blood cell recruitment

作　　者：Pavan Kadandale ;Joshua D. Stender ;Christopher K. Glass 等

出　　处：Proceedings of the National Academy of Sciences. 2010 ;107(23):10502-10507.doi:10.1073/pnas.0914168107

出版社：The National Academy of Sciences of the United States of America

文章 ID：200517768

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4.Retinoid X receptor α controls innate inflammatory responses through the up-regulation of chemokine expression

作　　者：Vanessa Núñez ;Daniel Alameda ;Daniel Rico 等

出　　处：Proceedings of the National Academy of Sciences. 2010 ;107(23):10626-10631.doi:10.1073/pnas.0913545107

出版社：The National Academy of Sciences of the United States of America

文章 ID：200517787

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5.Regulated subset of G1 growth-control genes in response to derepression by the Wnt pathway

作　　者：Sung Hee Baek ;Chrissa Kioussi ;Paola Briata 等

出　　处：Proceedings of the National Academy of Sciences. 2003 ;100(6):3245-3250.doi:10.1073/pnas.0330217100

出版社：The National Academy of Sciences of the United States of America

文章 ID：200563564

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6.PPARγ and PPARδ negatively regulate specific subsets of lipopolysaccharide and IFN-γ target genes in macrophages

作　　者：John S. Welch ;Mercedes Ricote ;Taro E. Akiyama 等

出　　处：Proceedings of the National Academy of Sciences. 2003 ;100(11):6712-6717.doi:10.1073/pnas.1031789100

出版社：The National Academy of Sciences of the United States of America

文章 ID：200588461

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7.Regulation of somatic growth by the p160 coactivator p/CIP

作　　者：Zhiyong Wang ;David W. Rose ;Ola Hermanson 等

出　　处：Proceedings of the National Academy of Sciences. 2000 ;97(25):13549-13554.doi:10.1073/pnas.260463097

出版社：The National Academy of Sciences of the United States of America

文章 ID：200556402

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8.Facilitated replacement of Kupffer cells expressing a paraoxonase-1 transgene is essential for ameliorating atherosclerosis in mice

作　　者：Gary Bradshaw ;Alejandra Gutierrez ;Jon H. Miyake 等

出　　处：Proceedings of the National Academy of Sciences. 2005 ;102(31):11029-11034.doi:10.1073/pnas.0502677102

出版社：The National Academy of Sciences of the United States of America

文章 ID：200523327

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9.Paradoxical effect on atherosclerosis of hormone-sensitive lipase overexpression in macrophages

作　　者：Jean-Louis Escary ;Henry A. Choy ;Karen Reue 等

出　　处：JLR Papers In Press. 1999 ;40(3):397-404.

出版社：American Society for Biochemistry and Molecular Biology

文章 ID：192688678

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10.PPAR- and LXR-dependent pathways controlling lipid metabolism and the development of atherosclerosis

作　　者：Andrew C. Li ;Christopher K. Glass

出　　处：JLR Papers In Press. 2004 ;45(12):2161-2173.doi:10.1194/jlr.R400010-JLR200

出版社：American Society for Biochemistry and Molecular Biology

文章 ID：192689878

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