1.Second messengers involved in the two processes of presynaptic facilitation that contribute to sensitization and dishabituation in Aplysia sensory neurons

作　　者：O Braha ;N Dale ;B Hochner 等

出　　处：Proceedings of the National Academy of Sciences. 1990 ;87(5):2040-2044.doi:10.1073/pnas.87.5.2040

出版社：The National Academy of Sciences of the United States of America

文章 ID：75179300

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2.Protein synthesis during acquisition of long-term facilitation is needed for the persistent loss of regulatory subunits of the Aplysia cAMP-dependent protein kinase.

作　　者：P J Bergold ;J D Sweatt ;I Winicov 等

出　　处：Proceedings of the National Academy of Sciences. 1990 ;87(10):3788-3791.doi:10.1073/pnas.87.10.3788

出版社：The National Academy of Sciences of the United States of America

文章 ID：75179659

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3.Overexpression of an Aplysia shaker K+ channel gene modifies the electrical properties and synaptic efficacy of identified Aplysia neurons.

作　　者：B K Kaang ;P J Pfaffinger ;S G Grant 等

出　　处：Proceedings of the National Academy of Sciences. 1992 ;89(3):1133-1137.doi:10.1073/pnas.89.3.1133

出版社：The National Academy of Sciences of the United States of America

文章 ID：75182108

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4.Hippocampal long-term depression and depotentiation are defective in mice carrying a targeted disruption of the gene encoding the RI beta subunit of cAMP-dependent protein kinase

作　　者：E P Brandon ;M Zhuo ;Y Y Huang 等

出　　处：Proceedings of the National Academy of Sciences. 1995 ;92(19):8851-8855.doi:10.1073/pnas.92.19.8851

出版社：The National Academy of Sciences of the United States of America

文章 ID：75357464

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5.Post-tetanic potentiation at an identified synapse in Aplysia is correlated with a Ca2+-activated K+ current in the presynaptic neuron: evidence for Ca2+ accumulation

作　　者：R Kretz ;E Shapiro ;E R Kandel

出　　处：Proceedings of the National Academy of Sciences. 1982 ;79(17):5430-5434.doi:10.1073/pnas.79.17.5430

出版社：The National Academy of Sciences of the United States of America

文章 ID：75186213

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6.Serotonin modulates a specific potassium current in the sensory neurons that show presynaptic facilitation in Aplysia

作　　者：M Klein ;J Camardo ;E R Kandel

出　　处：Proceedings of the National Academy of Sciences. 1982 ;79(18):5713-5717.doi:10.1073/pnas.79.18.5713

出版社：The National Academy of Sciences of the United States of America

文章 ID：75186269

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7.Classical conditioning in Aplysia californica

作　　者：E T Walters ;T J Carew ;E R Kandel

出　　处：Proceedings of the National Academy of Sciences. 1979 ;76(12):6675-6679.doi:10.1073/pnas.76.12.6675

出版社：The National Academy of Sciences of the United States of America

文章 ID：200595355

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8.Mechanism of calcium current modulation underlying presynaptic facilitation and behavioral sensitization in Aplysia

作　　者：M Klein ;E R Kandel

出　　处：Proceedings of the National Academy of Sciences. 1980 ;77(11):6912-6916.doi:10.1073/pnas.77.11.6912

出版社：The National Academy of Sciences of the United States of America

文章 ID：200596345

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9.Intracellular injection of t he catalytic subunit of cyclic AMP-dependent protein kinase simulates facilitation of transmitter release underlying behavioral sensitization in Aplysia

作　　者：V F Castellucci ;E R Kandel ;J H Schwartz 等

出　　处：Proceedings of the National Academy of Sciences. 1980 ;77(12):7492-7496.doi:10.1073/pnas.77.12.7492

出版社：The National Academy of Sciences of the United States of America

文章 ID：200596471

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10.Presynaptic inhibition in Aplysia involves a decrease in the Ca2+ current of the presynaptic neuron

作　　者：E Shapiro ;V F Castellucci ;E R Kandel

出　　处：Proceedings of the National Academy of Sciences. 1980 ;77(2):1185-1189.doi:10.1073/pnas.77.2.1185

出版社：The National Academy of Sciences of the United States of America

文章 ID：200596576

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	J H Schwartz	2篇
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	E P Brandon	1篇
	E T Walters	1篇
	F D Wilson	1篇
	G S McKnight	1篇
	I Winicov	1篇
	J Camardo	1篇
	J D Sweatt	1篇
	K A Burton	1篇
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	K A Karl	1篇
	K C Martin	1篇
	K R Weiss	1篇
	M A Colicos	1篇
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	M Zhuo	1篇
	N Dale	1篇
	O Braha	1篇
	P Greengard	1篇
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	P J Pfaffinger	1篇
	P K Dash	1篇
	R Kretz	1篇
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	S A Siegelbaum	1篇
	S G Grant	1篇
	T J Carew	1篇
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	E R Kandel	13篇
	M Klein	3篇
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