1.Amyloid fibrils activate B-1a lymphocytes to ameliorate inflammatory brain disease

作　　者：Michael Phillip Kurnellas ;Eliver Eid Bou Ghosn ;Jill M. Schartner 等

出　　处：Proceedings of the National Academy of Sciences. 2015 ;112(49):15016-15023.doi:10.1073/pnas.1521206112

出版社：The National Academy of Sciences of the United States of America

文章 ID：147520018

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2.The design, synthesis, and evaluation of molecules that enable or enhance cellular uptake: Peptoid molecular transporters

作　　者：Paul A. Wender ;Dennis J. Mitchell ;Kanaka Pattabiraman 等

出　　处：Proceedings of the National Academy of Sciences. 2000 ;97(24):13003-13008.doi:10.1073/pnas.97.24.13003

出版社：The National Academy of Sciences of the United States of America

文章 ID：200556307

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3.Definition of MHC and T cell receptor contacts in the HLA-DR4restricted immunodominant epitope in type II collagen and characterization of collagen-induced arthritis in HLA-DR4 and human CD4 transgenic mice

作　　者：Ellen Christina Andersson ;Bjarke Endel Hansen ;Helle Jacobsen 等

出　　处：Proceedings of the National Academy of Sciences. 1998 ;95(13):7574-7579.doi:10.1073/pnas.95.13.7574

出版社：The National Academy of Sciences of the United States of America

文章 ID：200602607

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4.Identification of a common immune regulatory pathway induced by small heat shock proteins, amyloid fibrils, and nicotine

作　　者：Jonathan B. Rothbard ;Jesse J. Rothbard ;Luis Soares 等

出　　处：Proceedings of the National Academy of Sciences. 2018 ;115(27):7081-7086.doi:10.1073/pnas.1804599115

出版社：The National Academy of Sciences of the United States of America

文章 ID：218258141

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