1.Mutational landscape of primary, metastatic, and recurrent ovarian cancer reveals c-MYC gains as potential target for BET inhibitors

作　　者：Charles Li ;Charles Li ;Elena Bonazzoli 等

出　　处：Proceedings of the National Academy of Sciences. 2019 ;116(2):619-624.doi:10.1073/pnas.1814027116

出版社：The National Academy of Sciences of the United States of America

文章 ID：225725561

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2.Structure, domain organization, and different conformational states of stem cell factor-induced intact KIT dimers

作　　者：Yarden Opatowsky ;Irit Lax ;Francisco Tomé 等

出　　处：Proceedings of the National Academy of Sciences. 2014 ;111(5):1772-1777.doi:10.1073/pnas.1323254111

出版社：The National Academy of Sciences of the United States of America

文章 ID：90127868

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3.Early and multiple origins of metastatic lineages within primary tumors

作　　者：Zi-Ming Zhao ;Bixiao Zhao ;Yalai Bai 等

出　　处：Proceedings of the National Academy of Sciences. 2016 ;113(8):2140-2145.doi:10.1073/pnas.1525677113

出版社：The National Academy of Sciences of the United States of America

文章 ID：168413724

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4.Loss of TRIM33 causes resistance to BET bromodomain inhibitors through MYC- and TGF-β–dependent mechanisms

作　　者：Xiarong Shi ;Valia T. Mihaylova ;Leena Kuruvilla 等

出　　处：Proceedings of the National Academy of Sciences. 2016 ;113(31):E4558-E4566.doi:10.1073/pnas.1608319113

出版社：The National Academy of Sciences of the United States of America

文章 ID：182290612

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5.Distinct cellular properties of oncogenic KIT receptor tyrosine kinase mutants enable alternative courses of cancer cell inhibition

作　　者：Xiarong Shi ;Leiliane P. Sousa ;Elizabeth M. Mandel-Bausch 等

出　　处：Proceedings of the National Academy of Sciences. 2016 ;113(33):E4784-E4793.doi:10.1073/pnas.1610179113

出版社：The National Academy of Sciences of the United States of America

文章 ID：182290734

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6.Mutational landscape of uterine and ovarian carcinosarcomas implicates histone genes in epithelial–mesenchymal transition

作　　者：Siming Zhao ;Stefania Bellone ;Salvatore Lopez 等

出　　处：Proceedings of the National Academy of Sciences. 2016 ;113(43):12238-12243.doi:10.1073/pnas.1614120113

出版社：The National Academy of Sciences of the United States of America

文章 ID：184627774

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7.Inhibition of ErbB3 by a monoclonal antibody that locks the extracellular domain in an inactive configuration

作　　者：Sangwon Lee ;Etienne B. Greenlee ;Joseph R. Amick 等

出　　处：Proceedings of the National Academy of Sciences. 2015 ;112(43):13225-13230.doi:10.1073/pnas.1518361112

出版社：The National Academy of Sciences of the United States of America

文章 ID：113233577

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8.The tethered configuration of the EGF receptor extracellular domain exerts only a limited control of receptor function

作　　者：Dawn Mattoon ;Peter Klein ;Mark A. Lemmon 等

出　　处：Proceedings of the National Academy of Sciences. 2004 ;101(4):923-928.doi:10.1073/pnas.0307286101

出版社：The National Academy of Sciences of the United States of America

文章 ID：75175295

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9.A structure-based model for ligand binding and dimerization of EGF receptors

作　　者：Peter Klein ;Dawn Mattoon ;Mark A. Lemmon 等

出　　处：Proceedings of the National Academy of Sciences. 2004 ;101(4):929-934.doi:10.1073/pnas.0307285101

出版社：The National Academy of Sciences of the United States of America

文章 ID：75175296

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10.Insights into the molecular basis for fibroblast growth factor receptor autoinhibition and ligand-binding promiscuity

作　　者：Shaun K. Olsen ;Omar A. Ibrahimi ;Angela Raucci 等

出　　处：Proceedings of the National Academy of Sciences. 2004 ;101(4):935-940.doi:10.1073/pnas.0307287101

出版社：The National Academy of Sciences of the United States of America

文章 ID：75175297

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