1.Small Molecule Analogues of the parasitic worm product ES-62 interact with the TIR domain of MyD88 to inhibit pro-inflammatory signalling

作　　者：Colin J. Suckling ;Shahabuddin Alam ;Mark A. Olson 等

出　　处：Scientific Reports. 2018 ;8(1):2123.doi:10.1038/s41598-018-20388-z

出版社：Springer Nature

文章 ID：230254422

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2.IL-33/ST2 signalling and crosstalk with FcεRI and TLR4 is targeted by the parasitic worm product, ES-62

作　　者：Dimity H. Ball ;Lamyaa Al-Riyami ;William Harnett 等

出　　处：Scientific Reports. 2018 ;8(1):4497.doi:10.1038/s41598-018-22716-9

出版社：Springer Nature

文章 ID：230251948

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3.Dendritic cells provide a therapeutic target for synthetic small molecule analogues of the parasitic worm product, ES-62

作　　者：Felicity E. Lumb ;James Doonan ;Kara S. Bell 等

出　　处：Scientific Reports. 2017 ;7(1).doi:10.1038/s41598-017-01651-1

出版社：Springer Nature

文章 ID：235884241

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4.Aggregation of the human high affinity immunoglobulin G receptor (FcγRI) activates both tyrosine kinase and G protein-coupled phosphoinositide 3-kinase isoforms

作　　者：Alirio J. Melendez ;David J. Gillooly ;Margaret M. Harnett 等

出　　处：Proceedings of the National Academy of Sciences. 1998 ;95(5):2169-2174.doi:10.1073/pnas.95.5.2169

出版社：The National Academy of Sciences of the United States of America

文章 ID：200604234

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5.The helminth product, ES-62 modulates dendritic cell responses by inducing the selective autophagolysosomal degradation of TLR-transducers, as exemplified by PKCδ

作　　者：Russell J. Eason ;Kara S. Bell ;Fraser A. Marshall 等

出　　处：Scientific Reports. 2016 ;6(1).doi:10.1038/srep37276

出版社：Springer Nature

文章 ID：235867778

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	Miguel A. Pineda	2篇
	Alirio J. Melendez	1篇
	Christina N. Steiger	1篇
	David J. Gillooly	1篇
	David T. Rodgers	1篇
	Dimity H. Ball	1篇
	Felicity E. Lumb	1篇
	Fraser A. Marshall	1篇
	James Doonan	1篇
	Janet M. Allen	1篇
	Kamal U. Saikh	1篇
	Mark A. Olson	1篇
	Marlene Corbet	1篇
	Russell J. Eason	1篇
	Shahabuddin Alam	1篇

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	Margaret M. Harnett	5篇
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