1.Targeted deletion of βIII spectrin impairs synaptogenesis and generates ataxic and seizure phenotypes

作　　者：Michael C. Stankewich ;Babette Gwynn ;Thomas Ardito 等

出　　处：Proceedings of the National Academy of Sciences. 2010 ;107(13):6022-6027.doi:10.1073/pnas.1001522107

出版社：The National Academy of Sciences of the United States of America

文章 ID：200517062

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2.A widely expressed βIII spectrin associated with Golgi and cytoplasmic vesicles

作　　者：Michael C. Stankewich ;William T. Tse ;Luanne L. Peters 等

出　　处：Proceedings of the National Academy of Sciences. 1998 ;95(24):14158-14163.doi:10.1073/pnas.95.24.14158

出版社：The National Academy of Sciences of the United States of America

文章 ID：200603718

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3.Publisher Correction: Age-dependent ataxia and neurodegeneration caused by an αII spectrin mutation with impaired regulation of its calpain sensitivity

作　　者：Arkadiusz Miazek ;Michał Zalas ;Joanna Skrzymowska 等

出　　处：Scientific Reports. 2021 ;11.doi:10.1038/s41598-021-97068-y

出版社：Springer Nature

文章 ID：1035388415

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4.Age-dependent ataxia and neurodegeneration caused by an αII spectrin mutation with impaired regulation of its calpain sensitivity

作　　者：Arkadiusz Miazek ;Michał Zalas ;Joanna Skrzymowska 等

出　　处：Scientific Reports. 2021 ;11.doi:10.1038/s41598-021-86470-1

出版社：Springer Nature

文章 ID：1031924518

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