1.Hippocampal deletion of NaV1.1 channels in mice causes thermal seizures and cognitive deficit characteristic of Dravet Syndrome

作　　者：Rachael E. Stein ;Rachael E. Stein ;Joshua S. Kaplan 等

出　　处：Proceedings of the National Academy of Sciences. 2019 ;116(33):16571-16576.doi:10.1073/pnas.1906833116

出版社：The National Academy of Sciences of the United States of America

文章 ID：239019003

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2.Calcium sensor regulation of the CaV2.1 Ca2+ channel contributes to short-term synaptic plasticity in hippocampal neurons

作　　者：Evanthia Nanou ;Jane M. Sullivan ;Todd Scheuer 等

出　　处：Proceedings of the National Academy of Sciences. 2016 ;113(4):1062-1067.doi:10.1073/pnas.1524636113

出版社：The National Academy of Sciences of the United States of America

文章 ID：150364878

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3.Altered short-term synaptic plasticity and reduced muscle strength in mice with impaired regulation of presynaptic CaV2.1 Ca2+ channels

作　　者：Evanthia Nanou ;Jin Yan ;Nicholas P. Whitehead 等

出　　处：Proceedings of the National Academy of Sciences. 2016 ;113(4):1068-1073.doi:10.1073/pnas.1524650113

出版社：The National Academy of Sciences of the United States of America

文章 ID：150364879

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4.KATP channel gain-of-function leads to increased myocardial L-type Ca2+ current and contractility in Cantu syndrome

作　　者：Mark D. Levin ;Gautam K. Singh ;Hai Xia Zhang 等

出　　处：Proceedings of the National Academy of Sciences. 2016 ;113(24):6773-6778.doi:10.1073/pnas.1606465113

出版社：The National Academy of Sciences of the United States of America

文章 ID：179204182

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5.Basal and β-adrenergic regulation of the cardiac calcium channel CaV1.2 requires phosphorylation of serine 1700

作　　者：Ying Fu ;Ruth E. Westenbroek ;Todd Scheuer 等

出　　处：Proceedings of the National Academy of Sciences. 2014 ;111(46):16598-16603.doi:10.1073/pnas.1419129111

出版社：The National Academy of Sciences of the United States of America

文章 ID：95043982

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6.IgGs from patients with amyotrophic lateral sclerosis and diabetes target CaVα2δ1 subunits impairing islet cell function and survival

作　　者：Yue Shi ;Kyoung Sun Park ;Seung Hyun Kim 等

出　　处：Proceedings of the National Academy of Sciences. 2019 ;116(52):26816-26822.doi:10.1073/pnas.1911956116

出版社：The National Academy of Sciences of the United States of America

文章 ID：243451980

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7.Subtype-selective reconstitution of synaptic transmission in sympathetic ganglion neurons by expression of exogenous calcium channels

作　　者：Sumiko Mochida ;Ruth E. Westenbroek ;Charles T. Yokoyama 等

出　　处：Proceedings of the National Academy of Sciences. 2003 ;100(5):2813-2818.doi:10.1073/pnas.262787299

出版社：The National Academy of Sciences of the United States of America

文章 ID：200563492

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8.Requirement for the synaptic protein interaction site for reconstitution of synaptic transmission by P/Q-type calcium channels

作　　者：Sumiko Mochida ;Ruth E. Westenbroek ;Charles T. Yokoyama 等

出　　处：Proceedings of the National Academy of Sciences. 2003 ;100(5):2819-2824.doi:10.1073/pnas.262787699

出版社：The National Academy of Sciences of the United States of America

文章 ID：200563493

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9.An unexpected requirement for brain-type sodium channels for control of heart rate in the mouse sinoatrial node

作　　者：Sebastian K. G. Maier ;Ruth E. Westenbroek ;T. T. Yamanushi 等

出　　处：Proceedings of the National Academy of Sciences. 2003 ;100(6):3507-3512.doi:10.1073/pnas.2627986100

出版社：The National Academy of Sciences of the United States of America

文章 ID：200563610

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10.Reduced sodium channel density, altered voltage dependence of inactivation, and increased susceptibility to seizures in mice lacking sodium channel β2-subunits

作　　者：Chunling Chen ;Vandana Bharucha ;Yuan Chen 等

出　　处：Proceedings of the National Academy of Sciences. 2002 ;99(26):17072-17077.doi:10.1073/pnas.212638099

出版社：The National Academy of Sciences of the United States of America

文章 ID：200565063

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