1.β-Arrestin inhibits NF-κB activity by means of its interaction with the NF-κB inhibitor IκBα

作　　者：D. Scott Witherow ;Tiffany Runyan Garrison ;William E. Miller 等

出　　处：Proceedings of the National Academy of Sciences. 2004 ;101(23):8603-8607.doi:10.1073/pnas.0402851101

出版社：The National Academy of Sciences of the United States of America

文章 ID：75175552

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2.ACTH Receptors in the Adrenal: Specific Binding of ACTH-125I and Its Relation to Adenyl Cyclase

作　　者：Robert J. Lefkowitz ;Jesse Roth ;William Pricer 等

出　　处：Proceedings of the National Academy of Sciences. 1970 ;65(3):745-752.doi:10.1073/pnas.65.3.745

出版社：The National Academy of Sciences of the United States of America

文章 ID：75316550

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3.A Fraction of the Ventricular Myocardium That Has the Specificity of the Cardiac Beta-Adrenergic Receptor

作　　者：Robert J. Lefkowitz ;Edgar Haber

出　　处：Proceedings of the National Academy of Sciences. 1971 ;68(8):1773-1777.doi:10.1073/pnas.68.8.1773

出版社：The National Academy of Sciences of the United States of America

文章 ID：75317506

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4.Identification of the Cardiac Beta-Adrenergic Receptor Protein: Solubilization and Purification by Affinity Chromatography

作　　者：Robert J. Lefkowitz ;Edgar Haber ;Donald O'Hara

出　　处：Proceedings of the National Academy of Sciences. 1972 ;69(10):2828-2832.doi:10.1073/pnas.69.10.2828

出版社：The National Academy of Sciences of the United States of America

文章 ID：75318457

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5.β-arrestin- but not G protein-mediated signaling by the “decoy” receptor CXCR7

作　　者：Sudarshan Rajagopal ;Jihee Kim ;Seungkirl Ahn 等

出　　处：Proceedings of the National Academy of Sciences. 2010 ;107(2):628-632.doi:10.1073/pnas.0912852107

出版社：The National Academy of Sciences of the United States of America

文章 ID：200517466

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6.β-Arrestin1 modulates lymphoid enhancer factor transcriptional activity through interaction with phosphorylated dishevelled proteins

作　　者：Wei Chen ;Liaoyuan A. Hu ;Mikhail V. Semenov 等

出　　处：Proceedings of the National Academy of Sciences. 2001 ;98(26):14889-14894.doi:10.1073/pnas.211572798

出版社：The National Academy of Sciences of the United States of America

文章 ID：200563947

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7.Defective lymphocyte chemotaxis in β-arrestin2- and GRK6-deficient mice

作　　者：Alan M. Fong ;Richard T. Premont ;Ricardo M. Richardson 等

出　　处：Proceedings of the National Academy of Sciences. 2002 ;99(11):7478-7483.doi:10.1073/pnas.112198299

出版社：The National Academy of Sciences of the United States of America

文章 ID：200564148

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8.Independent β-arrestin 2 and G protein-mediated pathways for angiotensin II activation of extracellular signal-regulated kinases 1 and 2

作　　者：Huijun Wei ;Seungkirl Ahn ;Sudha K. Shenoy 等

出　　处：Proceedings of the National Academy of Sciences. 2003 ;100(19):10782-10787.doi:10.1073/pnas.1834556100

出版社：The National Academy of Sciences of the United States of America

文章 ID：200589018

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9.The kinase Grk2 regulates Nedd4/Nedd4-2-dependent control of epithelial Na+ channels

作　　者：Anuwat Dinudom ;Andrew B. Fotia ;Robert J. Lefkowitz 等

出　　处：Proceedings of the National Academy of Sciences. 2004 ;101(32):11886-11890.doi:10.1073/pnas.0402178101

出版社：The National Academy of Sciences of the United States of America

文章 ID：200590768

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10.β-Arrestin-dependent signaling and trafficking of 7-transmembrane receptors is reciprocally regulated by the deubiquitinase USP33 and the E3 ligase Mdm2

作　　者：Sudha K. Shenoy ;Aalok S. Modi ;Arun K. Shukla 等

出　　处：Proceedings of the National Academy of Sciences. 2009 ;106(16):6650-6655.doi:10.1073/pnas.0901083106

出版社：The National Academy of Sciences of the United States of America

文章 ID：200591429

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